Producción Científica UPeU

URI permanente para esta comunidadhttps://cris.upeu.edu.pe/handle/123456789/1

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Autor/a: search.filters.author.Marcus PetermannFecha de finalización: 1999

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Mostrando 1 - 10 de 13
  • Publicacion
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    [Elastase activity in man and atherosclerosis].
    (1966-04-01)
    Marcus Petermann
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    T Pereda
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    E. del Campo
  • Publicacion
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    [Gastric secretion: technics to measure gastric acidity].
    (1968-04-01)
    Jorge E. Valenzuela
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    Marcus Petermann
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    I Cruzat
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  • Publicacion
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    [Effects of ethanol on pancreatic secretion stimulated by a gastrin-like pentapeptide (I.C.I. 50,123)].
    (1969-09-01)
    Jorge E. Valenzuela
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    Marcus Petermann
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    ABINAIR BERNARDES DA SILVA
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    F Lorca
  • Publicacion
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    [Effects of ethanol in nitrogen balance and protein utilization in normal subjects (author's transl)].
    (1977-06-01)
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    Amber L.S. Cheng
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    Alejandro Villa Gomez
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    Marcus Petermann
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    T Ségure
  • Publicacion
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  • Publicacion
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    [Dipeptidase activity and protein absorption in rats maintained on an alcoholic diet for 6 months].
    (1978-09-15)
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    Marcus Petermann
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    Carlos Fernández-del Castillo
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    Paola Aedo
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    H Iturriaga
    Rats treated with ethanol during 10 to 14 weeks, do not present alteration of protein absorption in spite of alcohol's inhibiting action on amino acid transport. In these animals, intestinal dypeptidase activity at 24 weeks was similar in the alcoholic rats and in the control group, which may possibly be the explanation for the normal protein absorption.
  • Publicacion
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    Glucose tolerance and the insulin response in recently drinking alcoholic patients: Possible effects of withdrawal
    (1986-03-01)
    H Iturriaga
    ;
    Melani R. Kelly
    ;
    Daniel Bunout
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    María Eugenia Pino
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    T Pereda
  • Publicacion
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    Nitrogen economy in alcoholic patients without liver disease
    (1987-07-01)
    Daniel Bunout
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    Marcus Petermann
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    G Barrera
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    H Iturriaga
  • Publicacion
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    Glucose Turnover Rate and Peripheral Insulin Sensitivity in Alcoholic Patients without Liver Damage
    (1989-01-01)
    Daniel Bunout
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    Marcus Petermann
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    M. Bravo
    ;
    Mary M. Kelly
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    S Hirsch
    Glucose intolerance is frequently found in alcoholic patients and an impaired insulin response has been documented in them. To look for alternative mechanisms that could explain this intolerance, a glucose turnover using tritiated glucose and an euglycemic glucose clamp were performed to measure the glucose production rate and peripheral insulin sensitivity, respectively. Two groups of recently abstinent chronic male alcoholic patients without evidence of liver damage were studied. The glucose turnover technique showed a higher basal glucose production rate in alcoholics, compared with normal volunteers (2.83 +/- 0.29 vs. 1.84 +/- 0.22 mg/kg/min); an intravenous ethanol load significantly increased this rate. The euglycemic glucose clamp did not show peripheral insulin resistance in alcoholics, compared with controls.
  • Publicacion
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    Nutritional support in hospitalized patients with alcoholic liver disease.
    (1989-09-01)
    Daniel Bunout
    ;
    V Aicardi
    ;
    Sandra Hirsch
    ;
    Marcus Petermann
    ;
    Matt Kelly
    The effects of a nutritional support in hospitalized patients with alcoholic cirrhosis and liver failure were studied in a controlled protocol. Thirty-six patients were included, 17 were randomly assigned to an experimental group and the rest to a control group. Experimentals received a diet aiming at 50 kcal (209 kJ)/kg bodyweight/d and 1.5 g protein/kg bodyweight/d (as proteins of high biological value). Controls received the standard diet prescribed by the attending physician. The severity of liver failure and the nutritional status on admission were similar in both groups. The measured energy intake in controls was 1813 +/- 121 kcal/d (7589 +/- 506 kJ/d) and 2707 +/- 71 kcal/d (1131 +/- 297 kJ/d) in experimentals (P less than 0.001). The protein intake in controls was 47 +/- 3.8 g/d and in experimentals 80 +/- 3 g/d (P less than 0.001). There were seven deaths during the study period (two experimentals and five controls). No differences were observed in the evolution of liver failure, hepatic encephalopathy or nutritional status between both study groups. It is concluded that a higher energy and protein intake in these patients does not have adverse effects and is associated with a non-significant reduction in mortality.